Robbins and Cotran Pathologic Basis of Disease

abnormal accumulation acid activation acute addition adhesion affected agents antibodies antigens apoptosis appear associated bacteria become binding blood body bone called cancer cause cells cellular changes Chapter chromosome chronic clinical collagen common complement complex contain cytokines cytoplasm damage death defects deficiency deposition described differentiation discussed disease disorders endothelial cells enzymes example expression extracellular FIGURE formation function gene genetic glucose growth factors healing host human immune important increased individuals induced infection inflammation inflammatory inherited initial injury involved known leading leukocytes levels liver loss lymphocytes lysosomal macrophages major mechanisms mediators membrane molecular molecules muscle mutations myocardial infarction necrosis neutrophils normal occur organs pathway patients plasma platelet present proliferation proteins reactions receptors reduced regulation release repair response result role severe signaling skin specific stem cells stimulate structure surface syndrome synthesis tion tissue tumor usually vascular vessels virus wound